This is a blood problem where blood doesn’t clot typically because of a strange low platelets count. This is additionally called immune thrombocytopenia. A low degree of platelets causes bleeding and bruising. Platelets are particles of blood that are formed in the bone marrow. Platelets help to stop bleeding at the site of injury by making a clot together.

On the off chance that the body doesn’t create enough platelets, blood doesn’t make clots at the site of injury. This condition causes small blue spots on the skin and bleeding in the body. Patients give pinpoint-sized red specks on the skin that are called petechiae. These petechiae seem like a rash. Other than this, purple or pale blue spots might show up. 

There are two types of ITP: 

Acute thrombocytopenic purpura

This typically influences little youngsters, ages 2 to 6 years of age. The indications might follow a viral ailment, like chickenpox. Acute ITP as a rule begins abruptly and the signs and symptoms normally vanish in under a half year, or sometimes within a few months. Treatment is not required more frequently. The problem starts when the bleeding is prolonged and platelets continue to down. Acute ITP is the most well-known type of disease.

Chronic thrombocytopenic purpura.

The beginning of the problem can occur at any stage in life, and the signs and symptoms can last at least a half year, quite a long while, or a lifetime. Grown-ups suffer more frequently than kids do, however it affects the younger ones too. Females have it more frequently than guys. Persistent ITP can repeat regularly and requires nonstop subsequent consultations from a hematologist. 

What is chronic ITP? 

ITP can become constant, which is when platelet counts are low for quite a while. Chronic ITP can cause severe complications whenever left untreated. Treatment is required for chronic ITP.  You might have to see another medical care provider who has some expertise in treating blood disorders, similar to a hematologist-oncologist. 

Remember that ITP isn’t malignant growth and it isn’t infectious. 

 What are platelets? 

Platelets are small cells in the blood that cluster together to farm coagulation and stop the bleeding when a vein is harmed due to any injury. For instance, when your body farms a scab, or clump, at the injured site, platelets are the main cells at that place to start the coagulation. Individuals with ITP have lower measures of platelets in their blood, which makes it harder to form a clot at the site of injury. 

What are the Sign and symptoms of ITP? 

At first, there are no signs and symptoms of ITP. However, when they happen it gives the following medical issue. 

• Bleeding from gums and nose 
• Blood in urine and stool 
• Heavy menstrual flow in females
• Swelling in joints 
• Pinpoint bleeding under the skin( Petechiae development) 
• Uncontrollable bleeding after any injury or medical procedure 

When to see a specialist? 

If you don’t have any signs and symptoms of bleeding and your platelet count is low than normal then, at that point, you don’t have to take stress a lot over treatment. On the off chance that if you bleed a lot after any small injury too, and you experience bleeding from the nose or gums, or pinpoint bleeding under the skin then, go visit a specialist right away. 

What are the reasons for ITP? 

It generally happens when the immune system starts to attack platelets assuming them as harmful foreign particles. In past, this condition was known as “IDIOPATHIC THROMBOCYTOPENIA” since its original cause was unknown. However, at present, we can think that it happens due to the mistaken attack of the immune system on platelets.

• In kids, this condition is a trigger because of viral contaminations like mumps and influenza.

• In grown-ups, this condition is set off because of diseases like HIV, H.pylori, and hepatitis.
• Autoimmune infections like Rheumatoid Arthritis, Systemic lupus erythematosus and grave’s sickness, and so forth

How is idiopathic thrombocytopenic purpura diagnosed? 

• Total clinical history and blood tests are enough to diagnose the ITP. You might have these tests:
• Complete blood count (CBC). An estimation of size, number, and formation of various platelets in a particular volume of blood (to find platelets count).
• Additional blood and urine tests. These tests are done to measure the bleeding and clotting time, including an additional blood test called an antiplatelet immunizer test. 
• A critical review of medications
• Bone marrow Aspiration: In the past, a bone marrow aspiration was thought to be enough for the finding of ITP. It may not be important with a positive antiplatelet immune response test, yet it is still normally done to take a general view of platelets production ITP when the antiplatelet antibody test is negative.

 What are the Complications of ITP? 

The most common complications of immune thrombocytopenia may include bleeding inside the brain that can be life-threatening. In females, heavy menstruation can cause anemia and other complications of anemia. 

What is the objective of treatment? 

The objective of treatment in ITP is to lessen the danger of bleeding by raising platelet counts. The required number of platelets will be transfused from one individual to another. The patient and the doctor decide the right treatment approach for the patient. Many people suffering from ITP try to change their lifestyles to limit the risk of bleeding.  

 What is the right platelet count? 

Each individual is unique. You and your medical care provider will figure out what the right platelet target is for you. Hematologists typically consider therapy when somebody with ITP has: Platelet counts under 20,000 for each microliter (μL) with Signs and symptoms of bleeding.

Treatment at Saher Homeo Clinic

Here, at Saher Welfare Foundation, we treat every individual at the genetic level. Every individual seeks treatment based on symptoms and health conditions. We have many completely cured cases. See their stories here     

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Anemia is a condition in which a person doesn’t have enough healthy red blood

cells to Convey sufficient oxygen to the tissues of the body. Feeling weakness and tiredness are key Symptoms of anemia. There are many types of anemia, each with its causation. Anemia can be short-term or long-term related to underline causes. Its intensity can be slighter to the extreme. See your physician immediately if you suspect that you are anemic, because it may be a warning sign. Treatment of

anemia is varied, as the type of anemia. It may be treated by supplements or

undergoing medical procedures. We can prevent anemia by consuming a healthy

diet.

What are the Signs & Symptoms of anemia?

Signs & symptoms of anemia may vary according to the underlying cause. They may include:

• Fatigue
• Weakness
•  Tiredness
•  Shortness of breath
• Yellowish or Pale skin
•  Chest pain
• Migraine

At the start, anemia can be mild to moderate that goes unnoticed for a long time. In any case, symptoms can worsen as anemia worsen.

When to see a specialist

Make a meeting with your specialist in case you’re feeling exhausted for unexplained reasons. A few anemias, for example, iron deficiency anemia or vitamin B-12 deficiency, are widespread.

Exhaustion has several causes other than anemia, so don’t panic that in case if you’re fatigued, it doesn’t mean you are anemic. A few people discover that their hemoglobin is low, which demonstrates anemia, at the time they go for blood donation. In case you’re informed that you can’t give blood as a result of low hemoglobin, make a meeting with your specialist.

What are the causes of Anemia?

An inadequate amount of red blood cells cause anemia. This may be due to:

•  The body doesn’t make sufficient RBCs
• Continues bleeding due to an injury or disease, lose RBCs more frequently than its formation.
• If the body destroys RBCs

The function of Red Blood Cells

The body makes three types of cells-Red blood cells to carry oxygen throughout the body, White blood cells to fight against infections, and platelets to help in blood clotting.

Red Blood Cells contain hemoglobin — an iron-rich protein that gives the red color to the blood. Hemoglobin empowers red blood cells to carry oxygen from your lungs to all cells of the body and to convey carbon dioxide from different cells of the body to your lungs so that it can be exhaled.

Most blood cells, including RBCs, are created normally in your bone marrow — a spongy material found inside the cavities of many of your large bones. To create hemoglobin and red blood cells, your body needs iron, nutrient B-12, folate, and different supplements from the foods you take.

Different types of anemia and their causes include:

Iron deficiency anemia:

This is the most well-known sort of anemia around the world. Iron deficiency anemia is brought about by a deficiency of iron in your body. Your bone marrow needs iron to make hemoglobin. Without sufficient iron, your body can’t create enough hemoglobin for red blood cells. Several pregnant women suffer from iron deficiency anemia. It is also caused by heavy menstrual flow, ulcers, cancers, and long-term use of pain relievers.

Causes of iron deficiency anemia in women:

There are many causes of iron deficiency anemia in women as:

• Very high menstrual flow (menorrhagia). This can be measured if she experiences heavy menstrual bleeding over several consecutive cycles. This heavy bleeding can cause an overall low level of blood and can trigger iron deficiency anemia.

· Pregnancy. At the time of pregnancy female’s body require extra iron to fulfill the needs of oxygen and nutrients to the baby. It is very common in our low socio-economic status where females don’t get enough neutrinos diet for themselves. Thus diversion of blood to the baby causes a low level of blood in the body of a female and as a result, she suffers from anemia.

Vitamin deficiency anemia:

Besides iron, the body also needs folate and Vitamin B-12 to produce healthy blood cells. If the diet lacks these nutrients, it can cause a decrees production of red blood cells. In some people, vitamin B-12 did not consume by the body. That may cause vitamin deficiency anemia, which is also called pernicious anemia.

Anemia due to chronic diseases:

Many diseases —, for example, malignant growth, HIV/AIDS, rheumatoid joint pain, kidney disease, Crohn’s disease, and other inflammatory diseases — can disturb the normal mechanism of RBCs production.

Aplastic Anemia:

This is a rare and health-threatening type of anemia. The body does not produce red blood cells according to the requirements. Frequent infections, autoimmune diseases, certain medicines, and exposure to toxic chemicals cause aplastic anemia.

Anemia due to bone marrow diseases:

Leukemia and myelofibrosis and many other diseases cause anemia by affecting RBCs production in the bone marrow. The impacts of these kinds of malignancy and disease are life-threatening.

Hemolytic Anemia: 

Destruction of red blood cells, more frequently than its production, causes hemolytic anemia. Hemolytic anemia can be inherited or can be developed at any stage of life.

Sickle cell Anemia: 

It is an inherited and most fatal form of anemia. This is caused by defective hemoglobin that forces red blood cells to form a crescent-like shape. These sickle-shaped cells die immaturely and cause a chronic lack of red blood cells.

Different Anemias:

There are many other types of anemias, for example, thalassemia and malarial anemia.

What are the complications of Anemia?

Anemia can cause serious complications if left untreated for a long time. The most serious complication is hypoxia of tissue, in which tissues of the body don’t get enough oxygen and tissue start to decay slowly. Low blood pressure, shock, pulmonary or coronary insufficiency may occur in severe cases.

Treatment at Saher Homeo Clinic

Here, we treat every individual at the genetic level. Every individual seeks treatment based on symptoms and health conditions. we have several completely cured cases. See their stories here

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What is Burkett’s Leukemia and lymphoma?

Burkett’s Leukemia is fast a growing and rare leukemia in which there is an excess of B-lymphocytes (white blood cells) in the blood and bone marrow. It usually starts into the lymph nodes as Burkett lymphoma, after that it spreads into the blood and bone marrow. which can grow directly into the blood and bone marrow, without the involvement of lymph nodes. Burkett’s leukemia and Burkett’s lymphoma are linked to the infection due to Epstein-Barr virus. Clinical presentation of Burkett’s leukemia is varied according to the site of origin and metastasized. Commonly it involves the gastrointestinal tract, head, and neck. Other primary sites can be abdomen,bone, liver, spleen, kidney, breast and pancreas.

How does Burkitt’s lymphoma affect the body?

Burkitt’s lymphoma presents with rapidly enlarging lymph node tumor masses involving the chest and/or the abdomen. It can spread into central nervous syetem and can involve spleen,liver and bone marrow. A number of other signs and symptoms can appear as night seating, sudden weight loss and fever.

Who does Burkitt’s lymphoma commonly affect?

Burkitt lymphoma stands almost only 1% of adult lymphoma, but mostly it can be appear as 30% of childhood non-Hodgkin lymphoma. Most children with Burkett’s lymphoma get diagnosed at the age of  5 and 10 years. While adults mostly get disgnosed between the ages of  30 to 50 years. Burkitt’s lymphoma effects males 4 times higher rates than females.

What are the Signs and symptoms of Burkett’s leukemia and lymphoma?

Sign and symptoms of Burkett’s leukemia may include the following In the case of abdominal BL, common sign and symptoms are

• Palpable mass
• Abdominal pain
• Nausea, Vomiting
• Constipation/Black stools
• Unexplained fever

When to visit the doctor?

If you feel any alarming symptoms that make you worry, visit your doctor immediately.

What are the Causes Burkett’s leukemia and lymphoma?

Causes of Burkitt’s leukemia are following

• Endemic Burkitt’s leukemia is linked to malaria and Epstein-Barr virus (EBV), that also causes glandular fever.
• The infrequent form of Burkitt’s leukemia is not as much of linked with the presence of Epstein- Barr virus (EBV) because in some cases people have this virus but they cannot develop Burkitt’s leukemia.
• People with immune-deficient syndromes like HIV/AIDS have higher chances to develop Burkitt’s leukemia than people without these syndromes.

What are the Types of Burkett’s leukemia and lymphoma?

According to the WHO criteria, there are 3 types of Burkitt’s leukemia.

Endemic BL:

The endemic form of Burkitt’s leukemia is most common in Africa. Almost 50% of childhood malignancies are of Burkitt’s leukemia. The mandibular, orbital bones and abdomen are the most common sites for the progression of endemic Burkitt’s leukemia. These types have more chances to spread into the central nervous system.

Sporadic BL:

It occurs worldwide equally. Almost 30-40% of children suffer from this leukemia. In the US and many European countries, it is diagnosed in the 40% pediatric lymphomas and 1-2% in adult leukemia cases. The abdomen is the most common site for the development of this form of BL.

Immunodeficiency-associated BL:

This type of BL is more common in patients with HIV/AIDS. It accounts for almost 30-40% of non-hodking lymphomas in the patients  of HIV and AIDS. It also occurs in the patients having congenital diseases that cause immune deficiency or in those who take immunosuppressive drugs for a long time.

RISK FACTORS

Infections:

Some viruses that directly affect the DNA of lymphocytes and can cause Burkitt’s leukemia such as a human T-cell lymphotropic virus (HTLV-1), Human herpesvirus 8 (HHV-8)  and Epstein-Barr virus (EBV).

Autoimmune disease:

Having Rheumatoid arthritis, Systemic lupus erythematosus and celiac disease are linked with the development of Burkitt’s leukemia.

Body weight and diet:

Being over-weight or obese have higher risks developing many health impairing issues including leukemias. Eating a healthy diet and staying in normal weight, can lower the risks of leukemias.

Radiation exposure:

Exposure to radiations for a long time can cause BL. Risks are higher for the people who got radiation therapy and chemotherapy for the treatment of cancer in the past.

Family history:

Having first-degree relatives who have cancer can increase your risks to suffer from Burkitt’s leukemia.

Race, geography:

It usually affects the people of Africa, the US, and Europe. In USA, white people are on higher risks than Asian American.

Diagnosis of Burkitt Lymphoma:

Occurrence and spread of Burkitt’s leukemia and  Lymphoma is very quick. So the prompt diagnosis is very important to prevent the disease. On suspicion of Burkitt Lymphoma, all or small part of enlarges lymph node is taken for biopsy. This biopsied tissue is examined under microscope to rule out the disease.

Additional tests may include:

• Ct scan of abdomen, chest and pelvis
• Chest X-ray
• Bone Marrow Aspiration/Biopsy
• PET SCAN
• Spinal fluid examination
• Blood tests to rule out liver and kidney functions
• Tests for HIV

What is Prognosis for Burkitt Lymphoma?

Burkitt lymphoma is deadly whenever left untreated. In youngsters, brief escalated chemotherapy typically fixes Burkitt lymphoma, prompting long term treatment prognosis is 60% to 90%. While in grown-up patients, results are more dynamic. Mostly, on time treatment is associated with long term treatment with the survival of 70% to 80 percent.

Outline of the Burkitt’s Leukemia and lymphoma:

Burkitt’s leukemia is Burkitt’s lymphoma yet with the predominance of dangerous Burkitt’s cells in the bone marrow up to 20%.

Burkitt’s lymphoma is a persuasive condition, and it is more common in kids than grown-ups. There are three kinds of Burkitt’s lymphoma: endemic, irregular, and immunodeficiency-related.

EBV, which is an transferable infection that normally spreads through salivation, has a solid relationship with a wide range of Burkitt’s lymphoma.

Diagnosis of Burkitt’s lymphoma and Burkitt’s leukemia might include biopsies and imaging tests, for example, CT outputs or MRI examines.

The best therapy choice for Burkitt’s lymphoma is thought repeated chemotherapy. Although this treatment technique is normally effective, it can bring about certain confusions and sideeffects, particularly in grown-ups.

Treatment at Saher Homeo Clinic

Here, at Saher Welfare Foundation, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. We have a number of completely cured cases.

The post Homeopathic Treatment of Burkett’s Leukemia and lymphoma appeared first on DR GHULAM YASEEN.

This type of leukemia is uncommon mostly, that develop in years and more slowly than acute myeloid leukemia. Chronic myeloid leukemia is also called chronic myelogenous leukemia or chronic granulocytic leukemia. It may affect mostly in older age, while In children its occurrence is rare.

Sign and symptoms

Chronic Myeloid Leukemia has following sign and symptoms

• Complete loss of appetite
• Frequent infections
• Fever
• Uneasiness with weakness
• Pale skin
• Excessive sweating during sleep
• Weight loss
• Easy bleeding

When to visit the doctor

Chronic myeloid leukemia may live unrevealed for many years in the first phase without any obvious sign and symptoms. Patients with chronic myeloid Leukemia have more chances of survival than Acute Myeloid Leukemia. So, it’s good to visit a doctor immediately if you experience persistent sign and symptoms of chronic myeloid leukemia.

What are the causes of CML?

Whenever there is a change in the genes, then you may experience blood disorders. In chronic myeloid leukemia, certain genes get mutations that develop leukemia in years. In human, cells develop and work dependent on the data contained in every cell’s chromosomes. Chromosomes are long parts of DNA in every cell. DNA is the substance that conveys our qualities, the guidelines for our cells to work. We resemble our parents since they are the source of our DNA. Our genes influence on our qualities because they carry the instruction for our cells to do every minor activity.

When a cell divides into two new cells, it keeps a copy of DNA into its chromosomes. This is a very convoluted procedure and errors can occur due to certain conditions.

There are some genes in DNA that control the cell division.

Oncogenes:

these genes promote cell division and cell growth.

Tumer suppresser genes:

these genes suppress cell division and cause cell death at the same time.

Formation of Philadelphia chromosome

Any changes in the DNA can turn on oncogenes and turn off tumor suppressor genes. Every human cell consists of 23 pairs of chromosomes. Chronic Myeloid Leukemia occurs when part of chromosome 9 goes towards the part of 22 and part to 22 chromosomes to the chromososme 9.this whole process is called translocation and it makes chromosome 22 shorter in size than normal-scientists named it Philadelphia chromosome. In all cases of acute myeloid Leukemia, Philadelphia chromosome is found.

Philadelphia chromosome form new genes

This Philadelphia chromosome forms a new gene named, BCR-ABL gene by combining genes from chromosome 9  to genes from chromosome 22. This abnormal gene contains guidelines to produce a protein called tyrosine kinase, which promotes cancer cells to grow out of control.

Formation of diseased blood cells

Blood cells originate from bone marrow in the immature form normally. These stem cells mature and specialized into various types of blood cells and then circulate in the body like red blood cells, white blood cells, and platelets. This is the normal phenomenon of blood cells production. In CML this process doesn’t work properly. Tyrosine kinase that was formed by abnormal gene BCR-ABL causes to the production of too many white blood cells.most of the time these white blood cells contain abnormal Philadelphia chromosome. Diseased white blood cells don’t die like in normal conditions. These cells build ups in form crowds of diseased white blood cells in the normal cells and start damaging the bone marrow.

What are the Risk factors?

• There are certain factors that may trigger chronic myeloid leukemia.
• Exposure to radiations
• Previous treatment for cancers
• Being male
• Old age

Complications of Chronic Myeloid Leukemia

Chronic myeloid leukemia can cause a number of complications.

• Excessive bleeding:

• in normal condition whenever body bleeds, platelets form a clot at the site of bleeding.in chronic myeloid leukemia, due to the shortage of these blood cells, blood doesn’t clot that causes excessive bleeding. Which is harmful and cause anemia.
• Pain: chronic myeloid leukemia can cause bone pain because bone marrow expands when diseased white blood cells build up in it.
• Weakness: when diseased white blood cells collect in the healthy blood cells than the number of red blood cells also decreased. At this level sign and symptoms of anemia may develop to make an alarming sign to visit a doctor.
• Splenomegaly(enlarged spleen): due to the extra production of blood cells, these blood cells are sent into the spleen for storage.this cause the swelling and enlargement of the spleen. Due to which sense of fullness develops even after taking a small number of meals.
• Frequent infections: white blood cells fight against infections at normal circumstances. But, in chronic myeloid leukemia due to the formation of diseased white blood cells body become unable to fight against any sort of infection.
• Death: if chronic myeloid leukemia cannot treat successfully on time, the patient can die.

Treatment at Saher Welfare Foundation

Here, at Saher Welfare Foundation, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. We have a number of completely cured cases. See their stories here

Make an appointment

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Your blood contains little cell particles named platelets. At whatever point you are harmed or cut a vein, and draining beginning from that harmed part, then, at that point, these platelets surge towards the spot of injury to quit bleeding. Platelets remain together and convey messages to different platelets to come at the site of injury. 

They structure an attachment at the bleeding point; because of which draining stops quickly. This is the typical strategy of blood thickening. Glanzmann Thrombasthenia is a platelets work problem in which blood doesn’t clump after a cut or injury. This is brought about by an anomaly in the qualities of glycoproteins IIb/IIIa. These qualities are fibrinogen receptors which help to adhere platelets together to quit dying. Because of the shortfall of these fibrinogen receptors, platelets don’t stay together and it turns out to be extremely challenging to prevent draining from a physical issue Site.

 In 1000,000 individuals, 1 individual is having this illness around the world. This illness is found in all kinds of people. Youngsters are analyzed from the get-go throughout everyday life, typically in the initial five years of life. Since in these years they experience extreme draining scenes from gums during getting teeth or losing child’s teeth. A few kids drain often than ordinary after inoculation. They might encounter epistaxis. 

WHAT ARE THE SIGN AND SYMPTOMS OF GLANZMANN THROMBASTHENIA? 

The most normal side effects of Glanzmann thrombasthenia are

 • Bleeding from nose and gums

 • Heavy menstruation in females

 • Excessive bleeding after trauma or surgery

 • Heavy menstruation in women

 • Bleeding from nose or gums 

• Easy bruises 

• Small purple spots on the skin due to capillary damage (petechiae) 

Rare symptoms of GT included the following: 

• Bleeding in the head and brain, that may cause headache, loss of consciousness, drowsiness, and pain in the neck. 

• Bleeding in the gastrointestinal tract may cause bloody vomiting or bloody stools.

 • Bleeding in the joints leaving swelling and immobility of joints. 

WHEN TO VISIT THE DOCTOR?

 If your child bleeds for a long time on slight injury or bleeds from the nose more often, without any cause then visit a doctor. If you see purple spots on your child’s body and If he bleeds a lot after vaccination or upon losing baby teeth, then it’s an emergency condition; you may hurry to visit a doctor.

 WHAT ARE THE CAUSES OF GLANZMAN THROMBASTHENIA? 

• Glanzmann thrombasthenia is an autosomal recessive disorder, which means that both parents may carry these abnormal genes but they may not have the disease.

 • Acquired Glanzmann Thrombasthenia is associated with lymphoproliferative and autoimmune diseases, characterized by impaired functions of platelets. Diagnosing Glanzmann’s Disease Your doctor may use the following simple blood tests to help diagnose Glanzmann’s disease: 

• platelet aggregation tests: to see how well your platelets clot

 • complete blood count: to determine the number of blood platelets you have

 • prothrombin time: to determine how long it takes for your blood to clot

 • partial thromboplastin time: another test to see how long it takes for your blood to clot Your doctor may also test some of your close relatives to check if they have Glanzmann’s disease or any of the genes that may contribute to the disorder.

 How Glanzmann Thrombasthenia affects clot retraction? 

Glanzmann’s thrombasthenia (GT) is a genetic platelet surface receptor disorder of GPIIb/IIIa (ITG αIIbβ3), either qualitative or quantitative, which results in faulty platelet aggregation and diminished clot retraction. Spontaneous mucocutaneous bleeding is common and can lead to fatal bleeding episodes. 

HOW WE CAN PREVENT IT? 

You can prevent your next generation from Glanzmann thrombasthenia, for that you need a genetic counselor before you marry. Women with GT should discuss menstrual bleeding and any sort of issues of planned pregnancy with the hematologist or gynecologist.

 WHAT ARE THE RISK FACTORS? 

To quit bleeding, when different estimations don’t work, then, at that point, you need to go for platelets transfusions. Platelets are gotten from 1-2 people. There are hazards for contaminations because of transfusions. Patients might show transfusions reaction making it difficult to continue transfusion. Because of delayed bleeding, you lose a gigantic measure of blood, that causes pallor. Thus, you need to care for your Hb level and general wellbeing if you have Glanzman’s thrombasthenia for better endurance.

 Living with Glanzmann’s Disease: 

Glanzmann’s disease is a long-term disorder with no cure. There are many dangers of continual bleeding such as chronic anemia, neurological or psychiatric problems, and possibly death if enough blood is lost. People with Glanzmann’s have to be very careful when they are injured and bleeding occurs. Women who have the condition may develop iron deficiency anemia during their menstrual cycles. If you begin to bruise easily or bleed for unknown reasons, you should talk with your doctor.

 It may mean the disease is becoming worse or that there’s another underlying condition that your doctor needs to diagnose. Patient education Patients should also be advised to avoid contact sports as well as the use of aspirin and non-steroidal anti-inflammatory drugs. To mitigate the risk of gingival bleeding, patients should maintain excellent oral hygiene and participate in regular dental visits. Women in particular, due to the consequences of menorrhagia, need monitoring for iron deficiency and should be placed on iron supplementation if necessary. Patients should be HLA typed at diagnosis and monitored for HLA and anti-platelet antibodies regularly. Patients should also be counseled that bleeding from a minor wound, the gingiva, or the nose may be able to be stopped by applying pressure, and/or by taking an oral anti-fibrinolytic, but to seek medical attention if bleeding persists.

 Management of epistaxis

Epistaxis or nasal bleeding is mostly seen as a manifestation in the patients suffering from GT. Sometimes a patient may respond to compression, nasal packings, and anti-fibrinolytic therapy, but these methods don’t work every time. Researchers believe that epistaxis in children can be stopped by the use of anterior and posterior packing and there may not be any need for prothrombotic agents 

TREATMENT AT SAHER homeo clinic

Here, at Saher Homeo Clinic, we treat every individual at the genetic level. Every individual seeks treatment based on symptoms and health conditions. We have several completely cured casesC

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MYELODYSPLASTIC SYNDROME is a group of disorders in which blood cells don’t form properly, immature blood cells in born marrow don’t mature; therefore normal healthy blood cells aren’t formed. It is commonly considered that there is no treatment for myelodysplastic syndrome. You can only reduce or manage bad health ailments. At some level, your doctor may suggest chemotherapy or bone marrow transplant.

Sign and symptoms

At early stages, you may not experience any sign and symptoms, but with the time you may experience following symptoms

• Unusual paleness of skin
• Shortness of breath
• Easy bruising
• Bleeding due to the low platelets count
• Pinpoint radish spots under the skin
• Tiredness
• Frequent infections

When to visit the doctor

Make an immediate appointment if you experience any sign and symptoms that make you worry.

Causes

In healthy individuals, the bone marrow makes immature blood cells that mature with the passage of time. In myelodysplastic syndrome something disturbs this normal procedure and more immature blood cells are formed. Formation of these defective cells leads to the low blood cells level in the body. Due to which sign and symptoms of anemia occur.

• Fatigue, tiredness and shortness of breath is caused by anemia,
• Infections are caused by leucopenia(decreased number of white blood cells)
• Bleeding caused by thrombocytopenia(low platelets count)

In some cases, the myelodysplastic syndrome hasn’t any known cause. Previous cancer treatments, exposure to the radiation and chemotherapy, constant use of certain drugs like benzene, heavy metals, etc.

Types of myelodysplastic syndromes

According to the criteria of WHO, MDS is divided into many subtypes according to the type of red blood cells, platelets, and white blood cells involvement. Types of Myelodysplastic syndrome include:

Ø  MDS with single lineage dysplasia (MDS-SLD):

This is a rare type of MDS that sometimes progress as acute myeloid leukemia. Otherwise, patients with this type of MDS remain can survive for a long time without getting any treatment. In this type one blood cell type is  abnormal (either red blood cells, white blood cells or platelets)

MDS with multilineage dysplasia (MDS-MLD):

This is the most common type of MDS also known as refractory cytopenia with multiple lineage dysplasia. In this type, more than 10% abnormality is seen in two or three types of cells in the bone marrow.

Ø  MDS with ring sideroblasts (MDS-RS)

In this type of   MDS, A number of early red blood cells are ring sideroblasts due to the mutations in the 5% of them. For diagnosis of this type, you must have 15% of red blood cells ring sideroblasts. This type is further divided into two subtypes

• MDS-RS with single lineage dysplasia (MDS-RS-SLD):

• In this type, only one type of cell is involved.

• MDS-RS with multilineage dysplasia (MDS-RS-MLD):

• Involvement of more than one cell type is seen in MDS-RS multilineage dysplasia.

This type is uncommon and it rarely develops in the acute myeloid leukemia. Patients with this type have better outcomes than any other MDS. In the past, it was referred to as anemia with ring sideroblasts.

Ø  MDS with excess blasts (MDS-EB)

In this type of MDS, there are the excess of blasts in the bone marrow than normal. The patient also has one type of blood cells involvement. There may be severe dysplasia in the bone marrow. This condition is also divided into two more subtypes.

• MDS-EB1:

Blasts makeups in the 5-9% of the cells in the bone marrow and 2% to 4% into the blood cells.

• MDS-EB2:

Blasts cells make-ups in the 10% to 19% of cells in the bone marrow and 55 to 19% cells in the blood. This type of MDS has higher chances to turn into the acute myeloid leukemia, higher for MDS-EB2 than MDS-EB1. Previously it was known as refractory anemia with excess blasts.

Ø  MDS associated with abnormal del chromosome:

People with this type have a very low number of red blood cells due to the missing of chromosome 5 in the chromosomes of bone marrow. Its reasons aren’t clear yet but it is considered the syndrome of older women. Its prognosis is good and the patient can survive for a long time and it rarely progress into AML.

Ø  Unclassified myelodysplastic syndrome:

This type is uncommon because its findings don’t fit in any other category of MDS. There are one from three types of blood cells are abnormal. Either white blood cells or platelets look abnormal under the microscope.

CLINICAL CLASSIFICATION OF MDS

Except for the classification of WHO, MDS is classified clinically into two types.

• Primary MDS:

If no cause can be identified.

• Secondary MDS:

When the cause of MDS is known. This is often termed after the known developed cancers.

Risk factors

Risk factors of MDS are following Old age: People after age of 60 years have higher chances to develop MDS. Previous treatment with chemotherapy or radiation therapy: Patients who receive treatment for cancers have higher chances to develop MDS. Exposure to certain chemicals: Exposure to certain drugs, tobacco, and heavy metals can lead to the progression of MDS. Genetic syndromes: People with inherited syndromes have higher risks to develop MDS.

• Diamond Blackfan anemia
• Fanconi anemia
• Familial platelet disorder with a propensity to myeloid malignancy
• Shwachman-Diamond syndrome
• Dyskeratosis congenital
• Severe congenital neutropenia

Complications of MDS may develop following complications with time

• Anemia: low count of red blood cells develop sign and symptoms of anemia
• Bleeding troubles: due to the low quantity of platelets, bleeding due to injury or surgery won’t stop.
• Risk of cancers: Certain types of MDS develop acute myeloid leukemia in later phases.
• Frequent infection: low numbers of white blood cells can cause frequent infections.

Treatment at Saher Homeo Clinic

Here, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. We have a number of completely cured cases. See their stories here     

The post Myelodysplastic Syndrome and Homeopathic Treatment appeared first on DR GHULAM YASEEN.

OVERVIEW

Microcytosis means the small size of RBCs as compared to the size of healthy RBCs, and anemia referred to the low number of red blood cells in the body. Collectively we can say that microcytic anemia is a blood deficiency disorder in which there are very few red blood cells of small size. There are certain factors that inhibit the production of hemoglobin, these factors or condition cause microcytic anemia.

Hemoglobin is an essential component of your blood, that carry oxygen to every cell of the body. More often iron deficiency causes microcytic anemia because your body needs iron for the production of hemoglobin. There are many types of microcytic anemia with different causation. Your doctor starts treatment according to the underlying cause of anemia.

According to world health organization almost 23% population of world is suffering from anemia from which more than 70% are suffering from iron deficiency anemia. Among the females, almost 41% females are suffering from anemia. While men rarely suffer with microcytic anemia due to circulating testosterone levels.

What are the Sign and symptoms of microcytic anemia?

At the start, you may not experience any signs and symptoms of microcytic anemia. When iron deficiency affects the tissues of the body, then signs and symptoms appear typically. Common signs and symptoms of microcytic anemia are the following:

• Dizziness
• Shortness of breath
• Pale skin
• Fatigue, weakness
• Headache
• Pain in body

When to visit the doctor?

If you feel sign and symptoms of anemia that do not resolve for two weeks then visit your doctor as soon as possible.

What are the Types of Microcytic Anemia?

Microcytic anemia can be further divided into three types according to the presence of hemoglobin levels. These are hypochromic, normochromic and hyperchromic anemia.

Hypochromic anemia:

In this type, there is a low hemoglobin level in the RBCs, which causes hypochromic microcytic anemia. Low hemoglobin level causes decrease oxygen supply to the tissues body-due to which skin turns its healthy color to paler. Further, in hypochromic microcytic anemia, there are very few red blood cells with small size. Hypochromic microcytic anemia has following causation Iron deficiency anemia: most common cause of microcytic anemia is iron deficiency. It may be due to the:

• Low increase intake of iron in your diet
• Certain health conditions, like celiac disease or halobacter pylori decrease the absorption of iron from the diet
• Chronic inflammatory bowel disease or ulcers in the gastrointestinal tract that cause frequent bleeding.
• Heavy menstruation in females
• Pregnancy

Sideroblastic anemia: 

A genetic mutation or acquired condition in which the body don’t form hemoglobin. Bone marrow produces ring-shaped sideroblastic rather than normal red blood cells. In this condition, iron builds up in the red blood cells.

Thalassemia:

A genetic disorder in which synthesis of faulty hemoglobin occurs due to the mutations in the hemoglobin forming genes.

 Normochromic Microcytic Anemias:

in normochromic anemia red blood cells have a normal hemoglobin level but you have a low quantity of them. Under the microscope you can view that their size is normal. Conditions in which you may have normochromic anemia:

Anemia of inflammation or chronic diseases:

This microcytic anemia may be seen in the patients who have

• Infectious disease like HIV/AIDS, tuberculosis, etc
• Inflammatory disease like rheumatoid arthritis, Crohn’s disease, or diabetes mellitus
• Kidney disease
• Cancers

Due to these conditions, absorption or utilization of iron decreased due to the abnormal functioning of red blood cells.

Hyperchromic  microcytic anemias:

Hyperchromic means that red blood cells have more hemoglobin than normal. This excess of hemoglobin makes red blood cells hue deep and more red in color than normal. These anemias are very rare.

Congenital spherocytic anemia:

This anemia is caused by a genetic condition which is also named as congenital spherocytic anemia. It is also called hereditary spherocytosis. In this condition, the outer membrane of your red blood cells has an imperfection in shape. That cause rigid and unorganized shape. They are sent in the spleen for break down because they cannot circulate in the blood.

Other  causes of Microcytic anemia:

• Excessive use of certain drugs
• Lead toxicity
• Deficiency of copper
• Excess of zinc

How Microcytic anemia diagnose?

When you feel sign and symptoms of the Microcytic anemia then your doctor asks for the complete blood count test. If your hemoglobin level shows up very low in it then another test named peripheral blood film performed for to see the shape and size of blood cells. This test can spot early detection of macrocytic or Microcytic changes in the red blood cells.

Once the diagnosis is made you may ask for further evaluation to see the cause of Microcytic anemia. As tests can be performed if you have celiac disease or any other infection inside your body.

What are the complications of Microcytic anemia?

Microcytic anemia due to deficiencies of  nutritional supplements and proper diet can be cured gradually with proper treatment. If it is due to any other underlying cause, then after the treatment of condition anemia cured on its own.

Anemia left untreated then, it can cause serious complications like

• Hypotension or low blood pressure
• Shock
• Coronary artery problems
• Pulmonary artery problems

How we can prevent Microcytic anemia with our diet?

Best way to prevent Microcytic anemia is to take proper amount of iron in your diet. A regular intake of calcium is also necessary for the absorption of iron. There are some diets that can help to prevent Microcytic anemia.

Foods that are rich in iron are:

• Red Meat
• Chicken
• Dark leafy vegetables
• Dry fruits/ nuts
• Beans

Food which is rich in vitamin C:

• Citrus fruits like oranges and grape fruits
• Milk, cheese and other dairy products
• Strawberries
• Broccoli

Treatment at Saher Homeo Clinic

Here, at Saher Homeo Clinic, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. We have a number of completely cured cases. See their stories here

The post Treatment of Microcytic Anemia in Homepathy appeared first on DR GHULAM YASEEN.

  Pure red cell aplasia is a very rare blood disorder in which the production of erythrocytes ceased due to the genetic mutation. It may occur in the patients that have a disease like tumors, systemic lupus erythematosus, thymomas, autoimmune diseases. PRCA has following three types usually.

What are the types of Pure red cell aplasia?

There are following types of pure red cell aplasia.

Congenital Pure Red Cell Aplasia:

This is also called diamond-blackfan anemia. It is usually diagnosed in the first two years of life, but in severe conditions, it may be diagnosed in first 2,3 months of life. Some children with pure red clel aplasia usually show signs and symptoms of malformation and some disabilities in mental level. Only several hundred children reported cases of congenital aplasia worldwide.

Acquired red cell aplasia:

This is a rare form of red cell aplasia in affecting adults. It is considered that acquired pure red cell aplasia is caused by any immune disorders, recurrent infections, certain medications and a tumor in the thymus gland.

Acute-self limiting pure red cell Aplasia:

This is a most common type of red cell aplasia caused by a certain drug or viral attack.most of the time when virus or medicine eliminate from the body, the PRCA also disappear. If you already have hemolytic anemia, then PRCA can be fatal for you. There is a difference in pure red cell aplasia and aplastic anemia. In aplastic anemia, all the RBCs, WBCs, and platelets become affected and in case of pure red cell aplasia only red blood cells line is affected, while the number of white blood cells and platelets remain in normal ranges.

Affected Populations

Acquired form of pure red cell aplasia is very rare and it affects males and females in equal proportions.

Relation of Pure red cell aplasia with Aplastic anemia:

There is a major difference between pure red cell aplasia and aplastic anemia. In pure red cell aplasia only red blood cells are affected. Whilst in aplastic anemia all the cells that bone marrow form get affected. They become lower in quantity.

What are the Sign and symptoms of pure red cell aplasia?

All patients of PRCA have anemia, its Sign and symptoms include:

• Fatigue
• Low count of red blood cells
• Dizziness
• Breathless
• Pale skin

Patients with Acquired PRCA may have other sign and symptoms too, due to the underlying disease.

When to visit a doctor?

If your child has paler complexion and showed sign and symptoms of anemia by birth, then immediately took him to the doctor. In acquired PRCA, patient experience this condition with other fatal health issues. It can be diagnosed with lab investigations, as the person visit the doctor.

What are the Causes of Pure red cell aplasia?

Mostly it is assumed that PRCA is caused by autoimmune disorders and mistaken attack of the body’s own immune cells on the blood-forming stem cells.

• Congenital
• Certain drugs
• Infections
• Autoimmune disease
• Idiopathic

How we can diagnose PRCA?

There are some tests that required for the diagnosis of pure red cell aplasia.

• Physical exam and history
• Blood tests:
  • A complete blood count with a differential that looks at the total number and type of blood cells in your blood
  • A reticulocyte count to determine blood levels of young red cells
  • A blood smear so your doctor can look at your blood under a microscope
• Bone marrow tests, which includes bone marrow aspiration and biopsy. With help of these tests doctors find out how bone marrow looks.

In such case patient should look a doctor who works on the bone marrow failures.

Life expectancy with PRCA

• Life expectancy with congenital pure red cell aplasia is limited.
• Patients having PRCA due to the idiopathic cause have a life expectancy of 1-2decades.
• The life span of the patients with Acquired PRCA depends upon the underlying disease.

What are the Risk factors?

Risk factors of PRCA include if you have :

• Thymomas (tumors of the thymus gland)
• Certain viruses like Parvovirus B-19
• Inherited genetic disorders
• Autoimmune diseases, like hepatitis, rheumatoid arthritis or having a clonal large population of lymphocytes that attack red blood cells precursors in the bone marrow.
• Treatment at Saher Homeo Clinic
• Here, at Saher Welfare Foundation, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. we have a number of completely cured cases. See their stories here    
• Related Disorders
• There are some other blood disorders who’s symptoms may look similar to the pure red cells aplasia. They are thought for the differential diagnosis
• In Aplastic Anemia there is a complete failure of bone marrow to form blood cells including rbcs, wbcs and platelets. Some other types of anemia can cause red blood cells destruction. The reason for aplastic anemia is uncertain and sometimes it is unknown. It can be happen after a radiotherapy or toxic reaction to some chemicals. In some cases aplastic anemia can appear with the tumor of thymus gland.
• Another very rare Blackfan-Diamond Anemia is a genetic blood disorder in which patient suffer from blood deficiency after birth. With the blood cell abnormalities patient also suffer with paleness, lethargy, weakness and abnormal physical appearance.
• Fanconi’s Anemia is another rare form of aplastic anemia in which patient suffer with bone marrow abnormalities. They usually present with abnormally small size of head, pigmented skin, and abnormal functions of sex organs. Its complications may include infections such as meningitis, pneumonia, excessive bleeding and leukemia. Other malignancies can also occur.

The post Treatment of Pure Red Cell Aplasia in Homeopathy appeared first on DR GHULAM YASEEN.

Acute Myeloid leukemia starts in the inner part of the bone-spongy material in the bones that produce blood cells. It moves into the blood and can spread into the other parts of the body involving lymph nodes, liver, and central nervous system. In the US every year 18,000 patients diagnosed with leukemia. More than 10,000 die due to cancer.

Acute Myeloid Leukemia is also known as Acute Myelogenous Leukemia, nonlymphocytic leukemia, myeloblastic leukemia, and acute granulocytic leukemia.

Sign and symptoms

Initially, sign and symptoms of acute myeloid leukemia may imitate with flu or other general health conditions. Sign and symptoms vary as the type of cells affected.

Sign and symptoms may include:

• Pain in bones
• Fever
• Lethargy and weakness
• Shortness of breath
• Pale skin
• Frequent infections
• Bleeding from nose or gums

When to visit the doctor

If you feel any unusual signs and symptoms that make you tense then visit the doctor immediately.

Causes

Damage to DNA in the bone marrow cause acute myeloid leukemia. Due to this damage production of blood cells goes wrong. Bone marrow starts producing immature blood cells that later develop in the leukemic cells. These abnormal cells are unable to perform normal functions and these cells buildups in the normal cells and make a crowd. This may be due to the following reasons.

• Genetic mutations
• Exposure to Radiation
• Chemotherapy drugs
• Other genetic disorders like Down’s syndrome
• Other blood disorders like myelodysplasia, myelofibrosis, polycythemia or thrombocythemia

Treatment at Saher Homeo Clinic

Here, at Saher Homeo clinic, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition.

Make an appointment

The post Acute Myeloid Leukemia(AML) appeared first on DR GHULAM YASEEN.

It is a hereditary blood disorder in which your body form less hemoglobin and very few red blood cells, as compared to the normal healthy individual. Hemoglobin is a blood protein that carries oxygen to the cells of the body. Deficiency of hemoglobin causes anemia and leaving you fatigued. If you have mild symptoms of anemia you may not need to worry for treatment. If you have severed type of anemia, then you have to go for blood transfusions, regularly.

What are the Sign and symptoms of Thalassemia?

Sign and symptoms of thalassemia may include the following:

• Weakness
• Fatigue
• Facial bone deformities
• Swellings in the body
• Growth retardation
• Shady Urine
• Pale and yellow complexion

Sign and symptoms depend upon the severity and type of thalassemia. Alpha-thalassemia, Thalassemia intermedia, Cooley anemia are the common types of thalassemia. In the severe type of thalassemia, sign and symptoms appear as the baby born. While in the case of others, children develop symptoms within the first two years of life.

What are the Causes of thalassemia?

A mutation in DNA, that makes hemoglobin, cause thalassemia. This mutated gene pass through generations. Thalassemia disrupts the normal production of hemoglobin and RBCs.With less hemoglobin and few red blood cells, you develop anemia and feel fatigued all the time.

When to see a doctor?

If you notice continuous decline health of your child, with any symptom of thalassemia, visit your child’s specialist immediately.

What are the types  of Thalassemia?

The severity of thalassemia depends upon the number of gene mutations.if you have the single gene mutation, you thalassemia isn’t severe. If these mutations involved a number of genes then, thalassemia is more severe. Hemoglobin molecules are composed of alpha and beta particle, the effect of mutations on these particles

Alpha-Thalassemia

There are four genes that are involved in the formation of the alpha-hemoglobin chain. You get two from each parent .if you inherit:

• 1 mutated gene, you may not have any sign and symptoms of thalassemia, but you are a carrier and you have an ability to pass it on, in your generation.
• 2 mutated genesindicate mild sign and symptoms of thalassemia. This is called alpha-Thalassemia.
• 3 mutated genes, specify moderate to severe sign and symptoms of thalassemia.
• 4 mutated genes, this condition is very rare. With this condition, life expectancy is rare-Fetuses born with severe anemia. They often die shortly after birth or require a blood transfusion, throughout life. Baby born with this condition has to receive a blood transfusion or undergo bone marrow transplant.

Thalassemia minor

There are two genes that make the beta hemoglobin chain. You get 1 from each of your parents. If you inherit

1 mutated gene, you may have the very mild sign and symptoms of thalassemia. This condition is called beta thalassemia or thalassemia minor.

2 mutated genes, this condition is called thalassemia major. Babies born with these 2 mutated genes are healthy often at the time of birth. But they develop sign and symptoms, moderate to severe in the first 2 years of life. A Milder form of thalassemia major is called thalassemia intermedia, with 2 mutated genes of beta hemoglobin chain.

Who is at risk to suffer from thalassemia?

Thalasemia is more common in some races as Italians, Greeks, and Africans and in Asians. Thalassemia is an inherited disorder which means it is passed from parents to their children.

How thalassemia can be diagnosed?

Thalassemia generally diagnosed at very early age. Children with thalassemia major, show sign and symptoms mostly first year of life. That may get worse if left unmanaged and can be life threatening. There are number of blood tests and evaluation for the diagnosis of thalassemia.

• A complete blood count test is usually helpful to see the hemoglobin level in the blood. One with very low hemoglobin can be evaluated by cbc test. It is the first test for a suspect of thalassemia. People with thalassemia have fewer red blood cells and low hb levels. Those with alpha or beta traits may have smaller red blood cells.
• Measuring reticulocyte count is also helpful for diagnosis. Reticulocytes are the young red blood cells. If their value is less than normal then it can be predicted that bone marrow is not working properly and does not form enough red blood cells.
• Measuring iron levels can indicate anemia. But in patients of thalassemia iron deficiency may not be a cause of anemia.
• Hemoglobin electrophoresis is test that diagnose beta thalassemia.
• Genetic testing is usually done to diagnose alpha thalassemia.

What is prognosis of thalassemia?

Patients with mild thalassemia can expect a normal life expectancy. Patients with moderate or severe form of thalassemia can have good survival but only with continues treatment. Heart disease with the iron overload is the main cause of death of patients of thalassemia. So its extremely important to control iron level. A bone marrow transplant is complete cure for thalassemia. Surgical procedures might be requiring for the skeletal deformities in the patients suffering from thalassemia.

What kind of care require for the patient of thalassemia?

If you suffer from thalassemia or a care taker of a thalassemia patient you need to follow the care plan for the patient. Frequent blood count test and iron level test must be done. Heart function tests and liver function tests should also performed annually. As frequent transfusions can higher the chances to get viral infections. So its necessary to check upon the liver as well.

Prevention

If you carry a thalassemia gene, you can prevent it. But you need counseling about thalassemia. When you think about having your children, first consult a genetic counselor. If you have thalassemia, you can fight with it. We’ll help you.

Treatment at Saher Homeo Clinic

Here, we treat every individual at the genetic level. Every individual seeks treatment on the basis of symptoms and health condition. We have a number of completely well managed cases.

Make an appointment

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